Thus, the study on autophagy is of great significance.

因此，对自吞噬的研究有重要意义。

Glucagon treatment of liver leads to increased autophagy.

用高血糖素处理肝脏，会导制自噬作用增加。

Autophagy appears to involve an active participation of the vacuolar membranes.

自体吞噬似乎与液泡膜的积极参加有关。

In tumour cells with defects in apoptosis, autophagy allows prolonged survival.

在凋亡缺陷的肿瘤细胞中，自噬延长了生存期。

There is a close relationship between autophagy and apoptosis during cell death.

自噬与凋亡在细胞死亡过程中的关系十分密切。

Mice without the genes needed for autophagy quickly develop Parkinson 's-like symptoms.

缺少这一基因的老鼠由于不能自吞噬很快也呈现出与帕金森症类似的症状。

This article summarizes the role of autophagy in Tumorigenesis, Metastasis and Therapy.

本文对自噬在肿瘤发生、转移与治疗中的作用进行综述。

Autophagy plays an important role in cleaning dust particles and the injured mitochondria.

自噬对于巨噬细胞清除尘粒和损伤的线粒体具有重要作用。

But it was thought that the brain was largely resistant to autophagy under these conditions.

但却指出大脑在这些条件下大体是抵抗自噬现象的。

This concept has now been evaluated using cells and organisms deficient in autophagy genes.

现在这个观念已被使用在细胞和生物机体的自噬基因缺陷之评估当中。

When the autophagy was blocked by the researchers, though, those hunger signals were blocked.

当研究人员阻断自噬过程后，然而，那些饥饿信号也被阻断了。

This review focuses on the processes of autophagy and its roles in cell survival and cell death.

本文对自噬的过程及其在细胞存活和死亡中的作用作一综述。

The role of autophagy-lysosome pathway in neurodegeneration associated with Parkinson's disease.

帕金森病相关神经变性中自噬溶酶体途径的作用。

Conclusion Receptor-mediated endocytosis of ConA-HRP is associated with autophagy and apoptosis.

结论受体介导内吞和自噬同凋亡之间存在一定的关系。

Autophagy reduces inflammation but at the same time permits the clearance of microbial infection.

自噬可减轻炎症反应，同时也允许清除导致感染的微生物。

Objective To evaluate the characteristics of autophagy in fibrotic and postoperative remnant liver.

目的了解肝纤维化和肝切除时同肝脏再生并行的自噬现象的表现和差别。

According to Li, autophagy is the only known process that gets rid of abnormal proteins within cells.

李敏表示，自吞噬是清除细胞内异常蛋白质的唯一途径。

This study shows that the autophagy pathway is markedly changed in vulnerable brain regions after TBI.

本研究显示，自噬途径在外伤性脑损伤的后的脑受伤区域内有明显的变化。

In the current study, they report on a mutant strain of P. gingivalis (PG0717) that does not induce autophagy.

在这篇刚报道的研究中，他们报道了一种不具有激活自噬作用的龈紫单胞菌突变株（PG0717）。

Future studies will determine whether and how manipulation of the autophagy pathway improves post-TBI recovery.

今后的研究将决定自噬途径是否及如何操控外伤性脑损伤后的恢复改善。

Eukaryotic cells use autophagy and the ubiquitin-proteasome system as their major protein degradation pathways.

真核细胞使用自噬和泛素蛋白酶体系统，作为它们主要的蛋白质降解通道。

There is a mutual regulation between apoptosis and autophagy in death signaling process mediated by mitochondria.

线粒体介导了死亡信号通路中自噬与凋亡的相互作用。

Autophagy is regulated by an intricate network of signaling cascades that have not yet been entirely disentangled.

通过一种尚未完全了解的级联信号的复杂网络(系统)，可以调节自噬。

Paradoxically, although autophagy is primarily a protective process for the cell, it can also play a role in cell death.

矛盾的是，尽管自噬主要对于细胞起一种保护过程中，它也在细胞死亡中起着一定的作用。

As part of this work, several recent studies have focused on autophagy in heart disease elicited by changes in hemodynamic load.

作为这项工作的一部分，几项近来的研究集中于通过改变血流动力学负荷来引出自溶作用在心脏病中作用。

Paradoxically autophagy defects are associated with increased tumorigenesis but the mechanism behind this has not been determined.

自相矛盾的是，自噬缺陷与肿瘤发生的呈正相关，但这背后的机制仍没有被明确。

The ultrastructural and biochemical results clearly show that the autophagy pathway is significantly activated in neurons after TBI.

超微结构与生化结果清楚的显示，TBI之后，自噬路径在神经元内被显著激活。

Autophagy is commonly observed in metazoan organisms during programmed cell death (PCD), but its function in dying cells has been unclear.

在后生动物有机体的细胞程序性死亡(PCD)中可以普遍观察到自噬现象。但它在垂死细胞的功能作用还不清楚。