Myosin, Light Chain Kinase, Clone: K36, Mab anti.

肌球蛋白，轻链激酶，克隆号：K36，单克隆抗体。

Phosphorylation of myosin promotes its binding to F-actin.

肌球蛋白的磷酸化促进其与F-肌动蛋白的结合；

No change was found in muscle myosin fiber-type composition.

肌球蛋白纤维类型组成没有发现有改变。

Cardiac myosin activation: will theory and practice coincide?

心肌肌球蛋白活化：理论与实践相一致吗？

Myosin motors can be attached to the surface of a glass slide.

肌球蛋白马达可附着在载玻片表面。

Myosin-II Regulatory Light Chain, Human Non-Muscle, recombinant.

肌球蛋白-II调节轻链， 人非肌肉型， 重组。

Method: Cardiac myosin was isolated and purified from fresh pig heart.

方法：从新鲜猪心中分离提纯心肌肌凝蛋白。

The cells were identified to be cardiomyocytes by expression of cardiac myosin heavy chain.

细胞心肌肌凝蛋白表达鉴定为心肌细胞。

Figure a myosin molecule walks along an actin filament through a cycle of structural changes.

一个肌球蛋白分子通过结构的周期性变化沿着一根肌动蛋白丝行走。

The two myosin heavy chains are important components of the contractile apparatus of the cardiomyocyte46.

两个肌球蛋白重链是心肌细胞的收缩装置的重要组分。

Expression of muscle myosin heavy chain-1 gene decreased and muscle ubiquitin gene expression increased in sepsis.

感染后肌肉肌纤蛋白基因表达下降，而肌肉泛素蛋白基因表达显著增强。

A muscle is a very large number of fibers close together, containing two different substances myosin and actomyosin.

肌肉是由极多的彼此紧帖的纤维所组成的，它含有两种不同的物质：肌球蛋白和肌动球蛋白。

The process occurs as myosin proteins attached to organelles push against microfilaments arrayed throughout the cell.

这个过程发生，当肌球蛋白附着在细胞器上，由细胞细胞上排列的微丝推动的时候。

Objectives: To study the changes of ventricular myosin light chain (VMLC) gene transcription in rheumatic heart disease (RHD).

目的：研究风湿性心脏病(风心病)心衰时心室肌中肌球蛋白轻链(VMLC)的变化。

One that churns out actin and myosin, which link up to form units that can expand and contract, will become a muscle cell. And so on.

而那种大量炮制肌动蛋白和肌凝蛋白的细胞，连接在一起从而形成可扩展及缩小的单元，这种细胞将成为肌肉细胞。如此类推。

The distribution of EOMs fibers and the expression of actin and myosin might play important roles in the mechanism of comtant strabismus;

眼外肌纤维减少和收缩蛋白表达减弱在共同性斜视的发病中可能起重要作用；

Cardiac myosin was extracted from normal human cardiac muscle. The specific activity of ATPase of cardiac myosin was assayed for demonstrating it.

从人心肌提取了肌凝蛋白，并检测了其ATP酶活力的稳定性。

Springer wanted to know whether these organisms, Dictyostelium discoideum, would continue to grow even when deprived of a crucial motor protein, myosin.

Springer想要了解的是，这些有机体，Dictyostelium discoideum，在被剥夺了一种名叫肌凝蛋白的关键的动力蛋白之后，还能不能继续生长。

To understand how LXR regulates LDLR, the authors examined transcriptional target genes of LXR and identified a gene that encodes a myosin interacting protein, IDOL.

为了了解LXR如何调节低密度脂蛋白受体，作者研究了LXR靶基因的转录，并确定了一个能编码肌球蛋白互动蛋白的基因，IDOL。

Animals were killed at two or six weeks after injury, and the muscles were analyzed for weight, cross-sectional area, myosin fiber-type composition, and fat content.

在损伤后2周或6周处死试验动物，分析肌肉的重量，横截面积，肌球蛋白纤维类型组成和脂肪含量。

It is suggested that with the development of myocardial hypertrophy in early stage of MI, adaptive changes in myosin ATPase activity and isoenzyme distribution occur.

结果提示：MI早期随着非梗塞心肌肥大的建立，肌球蛋白atp酶活性及其同功酶分布也发生了适应性的改变。

SDS-polyacrylamide gel electrophoresis indicated that SPI could enhance the intensity of myosin heavy chain (MHC) of bighead surimi gels while KG had no effect on it.

聚丙烯酰胺凝胶电泳表明，SPI可增强肌球蛋白重链带（MHC）的强度，而KG对MHC带的强度影响不大。

The results showed that coincident with the increases in myocardial fiber diameter on 9 days after MI, both myosin ATPase activity and V_1 isoenzyme amount were depressed.

结果表明，MI后第9天，在心肌细胞直径增大的同时，肌球蛋白atp酶活性下降，同功酶v_1含量下降。至MI后第21天，上述这种变化更加明显。

Aim: To detect effect of the different frequency of chronic electrical stimulation (CES) on myofibrillar isoform, myosin heavy chain (MHC) and metabolic enzyme activities.

目的：探讨不同频率慢性电刺激对膈肌肌纤维亚型、肌球蛋白重链(mhc)亚型和代谢酶活性的适应性变化的影响。

Thus, the tip of the leading process actively pulls the soma forward during neuronal migration through a myosin II-dependent forward F-actin flow along the leading process.

这项研究表明，迁移神经元中前导突起顶端通过肌球蛋白介导的肌动蛋白纤维流动活跃的拉动胞体向前运动。

The results suggest that Tet or Ena partially reduce the hydroxyproline content and elevate myosin ATPase activity of hypertrophied myocardium in renovascular hypertensive rats.

实验结果表明粉防己碱可部分逆转肾血管性高血压所致的大鼠左心室肥厚，降低心肌胶原含量，升高肌球蛋白ATP酶活性。